FAK Kinase Activity Is Required for the Progression of c-MET/β-Catenin-Driven Hepataocellular Carcinoma.

Na Shang; Maribel Arteaga; Ali Zaidi; Scott J. Cotler; Peter Breslin; Xianzhong Ding; Paul Kuo; Michael Nishimura; Jiwang Zhang; Wei Qiu

doi:10.3727/105221616X691604

FAK Kinase Activity Is Required for the Progression of c-MET/β-Catenin-Driven Hepataocellular Carcinoma.

Na Shang, Maribel Arteaga, Ali Zaidi, Scott J. Cotler, Peter Breslin, Xianzhong Ding, Paul Kuo, Michael Nishimura, Jiwang Zhang, Wei Qiu

Research output: Contribution to journal › Article › peer-review

Original language	American English
Journal	Gene Expression
Volume	17
Issue number	1
DOIs	https://doi.org/10.3727/105221616X691604
State	Published - Aug 31 2016

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Biology
Molecular Biology

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10.3727/105221616X691604License: Unspecified

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@article{f4d00d879ab342a5a2f7497a77522905,

title = "FAK Kinase Activity Is Required for the Progression of c-MET/β-Catenin-Driven Hepataocellular Carcinoma.",

author = "Na Shang and Maribel Arteaga and Ali Zaidi and Cotler, \{Scott J.\} and Peter Breslin and Xianzhong Ding and Paul Kuo and Michael Nishimura and Jiwang Zhang and Wei Qiu",

note = "There is an urgent need to develop new and more effective therapeutic strategies and agents to treat hepatocellular carcinoma (HCC). We have recently found that deletion of Fak in hepatocytes before tumors form inhibits tumor development and prolongs survival of animals in a c-Met (MET)/β-catenin (CAT)-driven HCC mouse model.",

year = "2016",

month = aug,

day = "31",

doi = "10.3727/105221616X691604",

language = "American English",

volume = "17",

journal = "Gene Expression",

number = "1",

}

TY - JOUR

T1 - FAK Kinase Activity Is Required for the Progression of c-MET/β-Catenin-Driven Hepataocellular Carcinoma.

AU - Shang, Na

AU - Arteaga, Maribel

AU - Zaidi, Ali

AU - Cotler, Scott J.

AU - Breslin, Peter

AU - Ding, Xianzhong

AU - Kuo, Paul

AU - Nishimura, Michael

AU - Zhang, Jiwang

AU - Qiu, Wei

N1 - There is an urgent need to develop new and more effective therapeutic strategies and agents to treat hepatocellular carcinoma (HCC). We have recently found that deletion of Fak in hepatocytes before tumors form inhibits tumor development and prolongs survival of animals in a c-Met (MET)/β-catenin (CAT)-driven HCC mouse model.

PY - 2016/8/31

Y1 - 2016/8/31

UR - https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5064945/

U2 - 10.3727/105221616X691604

DO - 10.3727/105221616X691604

M3 - Article

VL - 17

JO - Gene Expression

JF - Gene Expression

IS - 1

ER -

FAK Kinase Activity Is Required for the Progression of c-MET/β-Catenin-Driven Hepataocellular Carcinoma.

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